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3 edition of Airways responsiveness and atopy in the development of chronic lung disease found in the catalog.

Airways responsiveness and atopy in the development of chronic lung disease

Airways responsiveness and atopy in the development of chronic lung disease

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  • 29 Currently reading

Published by Raven Press in New York .
Written in English

    Subjects:
  • Lungs -- Diseases, Obstructive -- Pathophysiology.,
  • Lungs -- Diseases, Obstructive -- Immunological aspects.,
  • Asthma.,
  • Airway Obstruction.,
  • Hypersensitivity, Immediate.,
  • Lung Diseases, Obstructive -- etiology.,
  • Lung Diseases, Obstructive -- physiopathology.

  • Edition Notes

    Includes bibliographies and index.

    Statementeditors, Scott T. Weiss, David Sparrow.
    ContributionsWeiss, Scott T., Sparrow, David.
    Classifications
    LC ClassificationsRC776.O3 A35 1989
    The Physical Object
    Paginationx, 334 p. :
    Number of Pages334
    ID Numbers
    Open LibraryOL2185787M
    ISBN 100881675350
    LC Control Number89003767

    FRANK E. SPEIZER, IRA B. TAGER; EPIDEMIOLOGY OF CHRONIC MUCUS HYPERSECRETION AND OBSTRUCTIVE AIRWAYS DISEASE, Epidemiologic Reviews, Volume 1, Issue 1, 1 Janua We use cookies to enhance your experience on our continuing to use our website, you are agreeing to our use of cookies.   COPD, asthma, and cystic fibrosis (CF) are obstructive lung diseases with distinct pathophysiologies and clinical phenotypes. In this paper, we highlight recent advances in our understanding of relationships between clinical phenotypes, host inflammatory response, and lung microbiota in these diseases. Although COPD, asthma, and CF largely have distinct lung microbiota .

    Disease of the airways in chronic obstructive pulmonary disease. M.G. Cosio Piqueras, M.G. Cosio. #ERS Journals Ltd ABSTRACT: The pathological hallmarks of chronic obstructive pulmonary disease (COPD) are inflammation of the small airways (bronchiolitis) and destruction of lung parenchyma (emphysema). CHAPTER 6Obstructive airways disease Learning objectives To understand the aetiology and epidemiology of asthma To learn about the diagnosis and differential diagnosis of asthma To understand the management of acute and chronic asthma To recognise the risk factors for fatal asthma To understand the aetiology and epidemiology of chronic obstructive pulmonary disease (COPD) To .

      Introduction. For many years the natural history of chronic obstructive pulmonary disease (COPD) was seen to be more or less synonymous with the change in forced expiratory volume in 1 s (FEV 1) from early adulthood and throughout life. With this approach we have been able to identify risk factors for an excess decline in FEV 1 and this has been interpreted as ‘risk of COPD’. 3, 4 A.   There is substantial epidemiological evidence supporting the concept that respiratory syncytial virus (RSV) lower respiratory tract infection in infancy may be linked to the development of reactive airway disease (RAD) in childhood. However, much less is known concerning the mechanisms by which this self-limiting infection leads to airway dysfunction that persists long after the virus is.


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Airways responsiveness and atopy in the development of chronic lung disease Download PDF EPUB FB2

Airways responsiveness and atopy in the development of chronic lung disease. New York: Raven Press, © (OCoLC) Online version: Airways responsiveness and atopy in the development of chronic lung disease.

New York: Raven Press, © (OCoLC) Document Type: Book: All Authors / Contributors: Scott T Weiss; David Sparrow. postulated that atopy and airways hyper-responsiveness represent endogenous factors that predispose an individual to the development of chronic non-specific (or obstructive) lung disease.

This hypothesis has important clinical implications for the treatment of chronic obstructive lung disease. Airways responsiveness is related to airway by:   Many patients with chronic obstructive lung disease have an allergic history and most show increased airways responsiveness to histamine.

1 Orie and colleagues 1 postulated that atopy and airways hyper-responsiveness represent endogenous factors that predispose an individual to the development of chronic non-specific (or obstructive) lung disease.

This hypothesis has important clinical implications for the treatment of chronic obstructive lung by:   david sparrow, george o'connor, scott t.

weiss; the relation of airways responsiveness and atopy to the development of chronic obstructive lung disease, epidem We use cookies to enhance your experience on our continuing to Cited by: The relation of airways responsiveness and atopy to the development of chronic obstructive lung disease. Sparrow D(1), O'Connor G, Weiss ST.

Author information: (1)Normative Aging Study, Veterans Administration Outpatient Clinic, Boston, MA. PMID: [PubMed - indexed for MEDLINE] Publication Types:Cited by: Atopy and Airways Responsiveness in Chronic Obstructive Pulmonary Disease Scott T. Weiss, M.D., M.S.

This article has no abstract; the first words appear below. Nonspecific airway hyperresponsiveness and atopy have been postulated to be risk factors that predispose individuals to the development of chronic obstructive pulmonary disease.

The purpose of the present study was to examine the relationship of methacholine airway responsiveness and markers of atopy to rate of decline in lung function among men, age 40 to 79 yr, followed in the Normative Aging Study.

Cigarette smoking is recognized as the most important factor in the development of chronic airflow obstruction, yet only a minority of cigarette smokers develop clinical disease.

Ininterest focused on the role of increased levels of airways responsiveness and atopy as possible potentiating factors for the development of chronic airflow obstruction. Atopy. Atopy can be defined as an individual’s predisposition of developing an IgE and eosinophillic-mediated disease such as allergic asthma, atopic dermatitis and chronic allergic rhino-conjunctivitis, which are often concurrently associated within the same individual or as successive episodes.

From: Allergens and Respiratory Pollutants, Bronchial hyperresponsiveness (BHR) is a common finding in asthma 1 and has also been observed in patients with chronic obstructive airways disease (COPD). 2 Cross sectional studies have found significant associations between BHR and respiratory symptoms, including wheezing, cough and shortness of breath.

3– 5 Population based studies, including the first cross sectional Swiss study on. Asthma is characterised by increased bronchial responsiveness and the data from longitudinal studies suggest that long term respiratory morbidity is associated with the development and persistence of increased bronchial responsiveness.

A high proportion of these harmful effects — lung cancer and chronic obstructive pulmonary disease (COPD) — have a fatal outcome [1].

Lung cancer is the commonest form of cancer in the USA. The small airways were assessed in surgically resected lung tissue from patients — 39 with stage 0 (at risk), 39 with stage 1, 22 with stage 2, 16 with stage 3, and 43 with stage 4 (very.

More importantly, 30 to 40% of adult patients with CVA, unless adequately treated, may progress to classic asthma. CVA shares a number of pathophysiological features with classic asthma such as atopy, airway hyper-responsiveness, eosinophilic airway inflammation and various features of airway.

some patients, the development of chronic inflammation may be associated with permanent alterations in the airway structure—referred to as airway remodeling—that are not prevented by or fully responsive to currently available treatments (Holgate and Polosa ).

The pathological hallmarks of chronic obstructive pulmonary disease (COPD) are inflammation of the small airways (bronchiolitis) and destruction of lung parenchyma (emphysema). The functional consequence of these abnormalities is airflow limitation.

Airway abnormalities and emphysema interact in a complex fashion in the development of airflow limitation in COPD.

In an attempt to improve. Reactive airways dysfunction syndrome (RADS) is described as the development of respiratory symptoms in the minutes or hours after a single accidental inhalation of a high concentration of irritant gas, aerosol, vapor, or smoke; these initial symptoms are followed by asthma-like symptoms and airway hyperresponsiveness that persist for a.

Small Airway Diseases Small airway disease is defined as a pathologic condition in which the small conducting airways are affected either primarily or in addition to alveolar or interstitial lung changes.

For the pathologist, small airway disease has the same meaning as bronchiolitis, a nonspecific term used to describe inflammation of the membranous and respiratory. Brief Summary: From toto characterize the role of allergy and airways responsiveness in modifying growth of lung function in children and young adults in a community-based random population, the Childhood Respiratory Study in East Boston.

From toto examine the relationship of respiratory symptoms and illnesses, cigarette smoking, airways responsiveness, and markers of inflammation to growth and decline in lung function.

A subset of severe asthma patients has fixed airways obstruction, which is characterized by incomplete reversibility to bronchodilator challenge. We aimed to elucidate the factors associated with fixed airways obstruction in a cohort of patients with severe asthma in Singapore.

patients from the Singapore General Hospital-Severe Asthma Phenotype Study (SGH-SAPS) were screened. The small airways of the lungs are commonly affected in pediatric and adult asthma.

Small airways disease has been related to asthma control, severity, and risk of exacerbation. Diagnosis of small airways disease can be best made through evaluation of surgical lung specimens. Noninvasive techniques including spirometry, plethysmography, nitrogen washout, impulse oscillometry, and cross.Chronic obstructive pulmonary disease (COPD) is a type of obstructive lung disease characterized by long-term breathing problems and poor airflow.

The main symptoms include shortness of breath and cough with sputum production. COPD is a progressive disease, meaning it typically worsens over time. Eventually, everyday activities such as walking or getting dressed become difficult.We thank Carlo Barbetta and colleagues for their letter regarding the role of atopy in small airways disease (SAD).

We agree that atopy is a strong risk factor for asthma, but whether atopy itself is a risk factor for SAD remains unclear. In our cohort, atopy was identified in both participants with asthma and controls although the prevalence was higher among those with asthma ( [80%] of.